The Salk Institute authored and published a scientific article revealing that the SARS-CoV-2 spike protein is what’s actually causing vascular damage in covid patients and covid vaccine recipients, promoting the strokes, heart attacks, migraines, blood clots, and other harmful reactions that have already killed thousands of Americans (source: VAERS.hhs.gov).
All four covid vaccine brands currently in widespread use either inject patients with the spike protein or, via mRNA technology, instruct the patient’s own body to manufacture spike proteins and release them into their own blood. This is how they work.
The spike protein “damages cells” causes “vascular disease” even without a virus present.
In an article entitled, “The novel coronavirus’ spike protein plays additional key role in illness“, published on April 30th, 2021, the Salk Institute warns that, “Salk researchers and collaborators show how the protein damages cells, confirming COVID-19 as a primarily vascular disease.”
From that article:
Now, a major new study shows that the virus spike proteins (which behave very differently than those safely encoded by vaccines) also play a key role in the disease itself.
The paper, published on April 30, 2021, in Circulation Research, also shows conclusively that COVID-19 is a vascular disease, demonstrating exactly how the SARS-CoV-2 virus damages and attacks the vascular system on a cellular level.
“A lot of people think of it as a respiratory disease, but it’s really a vascular disease,” says Assistant Research Professor Uri Manor, who is co-senior author of the study. “That could explain why some people have strokes, and why some people have issues in other parts of the body. The commonality between them is that they all have vascular underpinnings.”
The team then replicated this process in the lab, exposing healthy endothelial cells (which line our arteries) to the spike protein. They showed that the spike protein damaged the cells by binding ACE2. This binding disrupted ACE2’s molecular signaling to mitochondria (organelles that generate energy for cells), causing the mitochondria to become damaged and fragmented.
Previous studies have shown a similar effect when cells were exposed to the SARSCoV-2 virus, but this is the first study to show that the damage occurs when cells are exposed to the spike protein on its own.
“If you remove the replicating capabilities of the virus, it still has a major damaging effect on the vascular cells, simply by virtue of its ability to bind to this ACE2 receptor,the S protein receptor, now famous thanks to COVID…Further studies with mutant spike proteins will also provide new insight towards the infectivity and severity of mutant SARS CoV-2 viruses.” – Uri Manor
The Salk Institute work refers to a paper published in Circulation Research: SARS-CoV-2 Spike Protein Impairs Endothelial Function via Downregulation of ACE 2.
This paper is the first to document the mechanism by which spike proteins can cause vascular destruction by binding to ACE2 receptors and inhibiting the function of cellular mitochondria.